1. L. Djousse, B. Knowlton, M. Hayden, E. W. Almqvist, R. Brinkman, C. Ross, R. Margolis, A. Rosenblatt, A. Durr, C. Dode, P. J. Morrison, A. Novelletto, M. Frontali, R. J. Trent, E. McCusker, E. Gomez-Tortosa, D. Mayo, R. Jones, A. Zanko, M. Nance, R. Abramson, O. Suchowersky, J. Paulsen, M. Harrison, Q. Yang, L. A. Cupples, J. F. Gusella, M. E. MacDonald, and R. H. Myers. Interaction of normal and expanded CAG repeat sizes influences age at onset of Huntington disease. Am J Med Genet A, 119(3):279-82, 2003. Note: P50ns016367/ns/nindsJournal ArticleResearch Support, Non-U.S. Gov'tResearch Support, U.S. Gov't, P.H.S.United States. [WWW] Keyword(s): Adolescent, Adult, Age of Onset, Aged, Aged, 80 and over, Child, Child, Preschool, Cohort Studies, Humans, Huntington Disease/*genetics, Middle Aged, New England, Probability, Survival Rate, *Trinucleotide Repeats.



2. C. Laurent, D. Niehaus, S. Bauche, D. F. Levinson, S. Soubigou, S. Pimstone, M. Hayden, I. Mbanga, R. Emsley, J. F. Deleuze, and J. Mallet. CAG repeat polymorphisms in KCNN3 (HSKCa3) and PPP2R2B show no association or linkage to schizophrenia. Am J Med Genet B Neuropsychiatr Genet, 116(1):45-50, 2003. Note: Journal ArticleResearch Support, Non-U.S. Gov'tUnited Statesthe official publication of the International Society of Psychiatric Genetics. [WWW] Keyword(s): Case-Control Studies, DNA/chemistry/genetics, DNA Mutational Analysis, Family Health, Genotype, Humans, Linkage Disequilibrium, Polymorphism, Genetic, Potassium Channels/*genetics, *Potassium Channels, Calcium-Activated, Schizophrenia/*genetics, Small-Conductance Calcium-Activated Potassium Channels, Spinocerebellar Ataxias/*genetics, Trinucleotide Repeats/*genetics.

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