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Publications of I. Rivals
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Articles in journal or book chapters
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J. Laffaire,
I. Rivals,
L. Dauphinot,
F. Pasteau,
R. Wehrle,
B. Larrat,
T. Vitalis,
R. X. Moldrich,
J. Rossier,
R. Sinkus,
Y. Herault,
I. Dusart,
and M. C. Potier.
Gene expression signature of cerebellar hypoplasia in a mouse model of Down syndrome during postnatal development.
BMC Genomics,
10:138,
2009.
Note: Journal ArticleResearch Support, Non-U.S. Gov'tEngland.
[WWW]
Keyword(s): Analysis of Variance,
Animals,
Animals,
Newborn,
Cerebellum/*abnormalities/growth & development/*metabolism,
Chromosomes,
Disease Models,
Animal,
Down Syndrome/*genetics/metabolism,
Gene Expression Profiling,
Gene Expression Regulation,
Developmental,
Male,
Mice,
Oligonucleotide Array Sequence Analysis,
Trisomy.
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E. Ait Yahya-Graison,
J. Aubert,
L. Dauphinot,
I. Rivals,
M. Prieur,
G. Golfier,
J. Rossier,
L. Personnaz,
N. Creau,
H. Blehaut,
S. Robin,
J. M. Delabar,
and M. C. Potier.
Classification of human chromosome 21 gene-expression variations in Down syndrome: impact on disease phenotypes.
Am J Hum Genet,
81(3):475-91,
2007.
Note: Journal ArticleResearch Support, Non-U.S. Gov'tUnited States.
[WWW]
Keyword(s): Base Sequence,
Chromosomes,
Human,
Pair 21/*genetics,
Down Syndrome/*genetics,
Female,
*Gene Expression,
Humans,
Male,
Molecular Sequence Data,
Oligonucleotide Array Sequence Analysis,
Phenotype,
Transcription,
Genetic,
*Variation (Genetics).
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I. Rivals,
L. Personnaz,
L. Taing,
and M. C. Potier.
Enrichment or depletion of a GO category within a class of genes: which test?.
Bioinformatics,
23(4):401-7,
2007.
Note: Evaluation StudiesJournal ArticleEngland.
[WWW]
Keyword(s): Data Interpretation,
Statistical,
Database Management Systems,
*Databases,
Protein,
Gene Expression Profiling/*methods,
Information Storage and Retrieval/*methods,
Multigene Family/*physiology,
Oligonucleotide Array Sequence Analysis/*methods,
Proteins/*classification/*metabolism.
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M. C. Potier,
I. Rivals,
G. Mercier,
L. Ettwiller,
R. X. Moldrich,
J. Laffaire,
L. Personnaz,
J. Rossier,
and L. Dauphinot.
Transcriptional disruptions in Down syndrome: a case study in the Ts1Cje mouse cerebellum during post-natal development.
J Neurochem,
97 Suppl 1:104-9,
2006.
Note: Journal ArticleResearch Support, Non-U.S. Gov'tEngland.
[WWW]
Keyword(s): Animals,
Cerebellum/*growth & development/*metabolism,
Disease Models,
Animal,
Down Syndrome/*genetics,
Gene Expression,
Gene Expression Regulation,
Developmental,
Growth/genetics,
Mice,
Mice,
Mutant Strains,
Oligonucleotide Array Sequence Analysis,
*Transcription,
Genetic,
Trisomy.
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L. Dauphinot,
R. Lyle,
I. Rivals,
M. T. Dang,
R. X. Moldrich,
G. Golfier,
L. Ettwiller,
K. Toyama,
J. Rossier,
L. Personnaz,
S. E. Antonarakis,
C. J. Epstein,
P. M. Sinet,
and M. C. Potier.
The cerebellar transcriptome during postnatal development of the Ts1Cje mouse, a segmental trisomy model for Down syndrome.
Hum Mol Genet,
14(3):373-84,
2005.
Note: HD-31498/HD/United States NICHDJournal ArticleResearch Support, N.I.H., ExtramuralResearch Support, Non-U.S. Gov'tResearch Support, U.S. Gov't, P.H.S.England.
[WWW]
Keyword(s): Animals,
Cell Differentiation,
Cerebellum/growth & development/*metabolism,
Disease Models,
Animal,
Down Syndrome/*genetics/metabolism,
Gene Dosage,
*Gene Expression Profiling,
Gene Expression Regulation,
Developmental,
Mice,
Mice,
Inbred C57BL,
Principal Component Analysis.
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Last modified: Thu May 6 18:23:28 2010
Author: schwartz.
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